2.  
   Gangrenous necrosis
    
   1.  
      This type of necrosis most often affects the lower extremities or bowel and is secondary to vascular occlusion.
   2.  
      When complicated by infective heterolysis and consequent liquefactive necrosis, gangrenous necrosis is called wet gangrene.
   3.  
      When characterized primarily by coagulative necrosis without liquefaction, gangrenous necrosis is called dry gangrene.
3.  
   Fibrinoid necrosis
    
   1.  
      This deposition of fibrin-like proteinaceous material in the arterial walls appears smudgy and acidophilic.
   2.  
      Fibrinoid necrosis is often associated with immune-mediated vascular damage.
4.  
   Fat necrosis occurs in two forms:
    
   1.  
      Traumatic fat necrosis, which occurs after a severe injury to tissue with high fat content, such as the breast
   2.  
      Enzymatic fat necrosis, which is a complication of acute hemorrhagic pancreatitis, a severe inflammatory disorder of the pancreas
       
      1.  
         Proteolytic and lipolytic pancreatic enzymes diffuse into inflamed tissue and literally digest the parenchyma.
      2.  
         Fatty acids liberated by the digestion of fat form calcium salts (saponification or soap formation).
      3.  
         Vessels are eroded, with resultant hemorrhage.

https://www.andreasastier.com/blog/the-different-types-of-necrosis-and-their-histological-identifications